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This over-activation depletes NAD + reserves, contributing to metabolic reprogramming that favors glycolysis over oxidative phosphorylation

By activating mitophagy, mitigating inflammation, and restoring cellular energetics, this strategy offers a novel paradigm in AP treatment

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In vitro studies have demonstrated that overexpression of Angiogenin, a RNase associated with familial ALS, activates NRF2 which protects astrocytes from oxidative stress (Hoang et al., 2019) and curcumin mediated NRF2 induction ameliorates mitochondrial dysfunction (Lu et al., 2012)

Neuronal GABA production and synaptic release was not affected by the knockdown of these genes, as evidenced by the spontaneous IPSC amplitude and frequency recorded from these cells (Fig